Exercise alleviates Parkinsonism: clinical and laboratory evidence
Identifieur interne : 000738 ( Main/Corpus ); précédent : 000737; suivant : 000739Exercise alleviates Parkinsonism: clinical and laboratory evidence
Auteurs : T. Archer ; A. Fredriksson ; B. JohanssonSource :
- Acta Neurologica Scandinavica [ 0001-6314 ] ; 2011-02.
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Abstract
Archer T, Fredriksson A, Johansson B. Exercise alleviates Parkinsonism: clinical and laboratory evidence.
Acta Neurol Scand: 2011: 123: 73–84.
© 2010 John Wiley & Sons A/S. The present review examines the putative benefits for individuals afflicted with Parkinsonism, whether in the clinical setting or in the animal laboratory, accruing from different exercise regimes. The tendency for patients with Parkinson’s disease (PD) to express either normal or reduced exercise capacity appears regulated by factors such as fatigue, quality‐of‐life and disorder severity. The associations between physical exercise and risk for PD, the effects of exercise on idiopathic Parkinsonism and quality‐of‐life, the effects of exercise on animal laboratory models of Parkinsonism and dopamine (DA) loss following neurotoxic insults, and the effects of exercise on the DA precursor, l‐Dopa, efficacy are examined. It would appear to be case that in view of the particular responsiveness of the dopaminergic neurons to exercise, the principle of ‘use it or lose’ may be of special applicability among PD patients.
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DOI: 10.1111/j.1600-0404.2010.01360.x
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ISTEX:64987335DC08043EBFF4362DAEAA5CCDD0E660FELe document en format XML
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Acta Neurol Scand: 2011: 123: 73–84.
© 2010 John Wiley & Sons A/S. The present review examines the putative benefits for individuals afflicted with Parkinsonism, whether in the clinical setting or in the animal laboratory, accruing from different exercise regimes. The tendency for patients with Parkinson’s disease (PD) to express either normal or reduced exercise capacity appears regulated by factors such as fatigue, quality‐of‐life and disorder severity. The associations between physical exercise and risk for PD, the effects of exercise on idiopathic Parkinsonism and quality‐of‐life, the effects of exercise on animal laboratory models of Parkinsonism and dopamine (DA) loss following neurotoxic insults, and the effects of exercise on the DA precursor, l‐Dopa, efficacy are examined. It would appear to be case that in view of the particular responsiveness of the dopaminergic neurons to exercise, the principle of ‘use it or lose’ may be of special applicability among PD patients.</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>T. Archer</name><affiliations><json:string>Department of Psychology, University of Gothenburg, Gothenburg, Sweden</json:string></affiliations></json:item><json:item><name>A. Fredriksson</name><affiliations><json:string>Department of Neuroscience Psychiatry Ulleråker, University Hospital, Uppsala, Sweden</json:string></affiliations></json:item><json:item><name>B. Johansson</name><affiliations><json:string>Department of Psychology, University of Gothenburg, Gothenburg, Sweden</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>Parkinsonism</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>exercise</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>quality‐of‐life</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>function</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>dopamine</value></json:item></subject><articleId><json:string>ANE1360</json:string></articleId><language><json:string>eng</json:string></language><abstract>Archer T, Fredriksson A, Johansson B. Exercise alleviates Parkinsonism: clinical and laboratory evidence.
Acta Neurol Scand: 2011: 123: 73–84.
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Acta Neurol Scand: 2011: 123: 73–84.
© 2010 John Wiley & Sons A/S. The present review examines the putative benefits for individuals afflicted with Parkinsonism, whether in the clinical setting or in the animal laboratory, accruing from different exercise regimes. The tendency for patients with Parkinson’s disease (PD) to express either normal or reduced exercise capacity appears regulated by factors such as fatigue, quality‐of‐life and disorder severity. The associations between physical exercise and risk for PD, the effects of exercise on idiopathic Parkinsonism and quality‐of‐life, the effects of exercise on animal laboratory models of Parkinsonism and dopamine (DA) loss following neurotoxic insults, and the effects of exercise on the DA precursor, l‐Dopa, efficacy are examined. It would appear to be case that in view of the particular responsiveness of the dopaminergic neurons to exercise, the principle of ‘use it or lose’ may be of special applicability among PD patients.</p></abstract><textClass xml:lang="en"><keywords scheme="keyword"><list><head>Keywords</head><item><term>Parkinsonism</term></item><item><term>exercise</term></item><item><term>quality‐of‐life</term></item><item><term>function</term></item><item><term>dopamine</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="2011-02">Published</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><original>false</original><mimetype>text/plain</mimetype><extension>txt</extension><uri>https://api.istex.fr/document/64987335DC08043EBFF4362DAEAA5CCDD0E660FE/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Blackwell Publishing Ltd</publisherName><publisherLoc>Oxford, UK</publisherLoc></publisherInfo><doi origin="wiley" registered="yes">10.1111/(ISSN)1600-0404</doi><issn type="print">0001-6314</issn><issn type="electronic">1600-0404</issn><idGroup><id type="product" value="ANE"></id><id type="publisherDivision" value="ST"></id></idGroup><titleGroup><title type="main" sort="ACTA NEUROLOGICA SCANDINAVICA">Acta Neurologica Scandinavica</title></titleGroup></publicationMeta><publicationMeta level="part" position="02102"><doi origin="wiley">10.1111/ane.2010.123.issue-2</doi><numberingGroup><numbering type="journalVolume" number="123">123</numbering><numbering type="journalIssue" number="2">2</numbering></numberingGroup><coverDate startDate="2011-02">February 2011</coverDate></publicationMeta><publicationMeta level="unit" type="reviewArticle" position="1" status="forIssue"><doi origin="wiley">10.1111/j.1600-0404.2010.01360.x</doi><idGroup><id type="unit" value="ANE1360"></id></idGroup><countGroup><count type="pageTotal" number="12"></count></countGroup><titleGroup><title type="tocHeading1"><i>Review articles</i></title></titleGroup><copyright>© 2010 John Wiley & Sons A/S</copyright><eventGroup><event type="firstOnline" date="2011-01-04"></event><event type="publishedOnlineAcceptedOrEarlyUnpaginated" date="2010-05-17"></event><event type="xmlConverted" agent="Converter:BPG_TO_WML3G version:2.4.2 mode:FullText" date="2011-01-03"></event><event type="publishedOnlineFinalForm" date="2011-01-04"></event><event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:4.0.1" date="2014-03-15"></event><event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.1.7 mode:FullText,remove_FC" date="2014-10-14"></event></eventGroup><numberingGroup><numbering type="pageFirst" number="73">73</numbering><numbering type="pageLast" number="84">84</numbering></numberingGroup><correspondenceTo>Trevor Archer, Department of Psychology, University of Gothenburg, Box 500, SE‐430 50 Gothenburg, Sweden
Tel.: +4631 7864694
Fax: +4631 7864628
e‐mail: <email>trevor.archer@psy.gu.se</email></correspondenceTo><linkGroup><link type="toTypesetVersion" href="file:ANE.ANE1360.pdf"></link></linkGroup></publicationMeta><contentMeta><unparsedEditorialHistory>Accepted for publication March 3, 2010</unparsedEditorialHistory><countGroup><count type="figureTotal" number="0"></count><count type="tableTotal" number="0"></count></countGroup><titleGroup><title type="main">Exercise alleviates Parkinsonism: clinical and laboratory evidence</title><title type="shortAuthors"><b>Archer et al.</b></title><title type="short"><b>Exercise alleviates Parkinsonism</b></title></titleGroup><creators><creator creatorRole="author" xml:id="cr1" affiliationRef="#a1"><personName><givenNames>T.</givenNames><familyName>Archer</familyName></personName></creator><creator creatorRole="author" xml:id="cr2" affiliationRef="#a2"><personName><givenNames>A.</givenNames><familyName>Fredriksson</familyName></personName></creator><creator creatorRole="author" xml:id="cr3" affiliationRef="#a1"><personName><givenNames>B.</givenNames><familyName>Johansson</familyName></personName></creator></creators><affiliationGroup><affiliation xml:id="a1" countryCode="SE"><unparsedAffiliation>Department of Psychology, University of Gothenburg, Gothenburg, Sweden</unparsedAffiliation></affiliation><affiliation xml:id="a2" countryCode="SE"><unparsedAffiliation>Department of Neuroscience Psychiatry Ulleråker, University Hospital, Uppsala, Sweden</unparsedAffiliation></affiliation></affiliationGroup><keywordGroup xml:lang="en"><keyword xml:id="k1">Parkinsonism</keyword><keyword xml:id="k2">exercise</keyword><keyword xml:id="k3">quality‐of‐life</keyword><keyword xml:id="k4">function</keyword><keyword xml:id="k5">dopamine</keyword></keywordGroup><abstractGroup><abstract type="main" xml:lang="en"><p>Archer T, Fredriksson A, Johansson B. Exercise alleviates Parkinsonism: clinical and laboratory evidence.
Acta Neurol Scand: 2011: 123: 73–84.
© 2010 John Wiley & Sons A/S.</p><p>The present review examines the putative benefits for individuals afflicted with Parkinsonism, whether in the clinical setting or in the animal laboratory, accruing from different exercise regimes. The tendency for patients with Parkinson’s disease (PD) to express either normal or reduced exercise capacity appears regulated by factors such as fatigue, quality‐of‐life and disorder severity. The associations between physical exercise and risk for PD, the effects of exercise on idiopathic Parkinsonism and quality‐of‐life, the effects of exercise on animal laboratory models of Parkinsonism and dopamine (DA) loss following neurotoxic insults, and the effects of exercise on the DA precursor, <sc>l</sc>‐Dopa, efficacy are examined. It would appear to be case that in view of the particular responsiveness of the dopaminergic neurons to exercise, the principle of ‘use it or lose’ may be of special applicability among PD patients.</p></abstract></abstractGroup></contentMeta></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Exercise alleviates Parkinsonism: clinical and laboratory evidence</title></titleInfo><titleInfo type="abbreviated"><title>Exercise alleviates Parkinsonism</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Exercise alleviates Parkinsonism: clinical and laboratory evidence</title></titleInfo><name type="personal"><namePart type="given">T.</namePart><namePart type="family">Archer</namePart><affiliation>Department of Psychology, University of Gothenburg, Gothenburg, Sweden</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">A.</namePart><namePart type="family">Fredriksson</namePart><affiliation>Department of Neuroscience Psychiatry Ulleråker, University Hospital, Uppsala, Sweden</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">B.</namePart><namePart type="family">Johansson</namePart><affiliation>Department of Psychology, University of Gothenburg, Gothenburg, Sweden</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="review-article" displayLabel="reviewArticle"></genre><originInfo><publisher>Blackwell Publishing Ltd</publisher><place><placeTerm type="text">Oxford, UK</placeTerm></place><dateIssued encoding="w3cdtf">2011-02</dateIssued><edition>Accepted for publication March 3, 2010</edition><copyrightDate encoding="w3cdtf">2011</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType></physicalDescription><abstract lang="en">Archer T, Fredriksson A, Johansson B. Exercise alleviates Parkinsonism: clinical and laboratory evidence.
Acta Neurol Scand: 2011: 123: 73–84.
© 2010 John Wiley & Sons A/S. The present review examines the putative benefits for individuals afflicted with Parkinsonism, whether in the clinical setting or in the animal laboratory, accruing from different exercise regimes. The tendency for patients with Parkinson’s disease (PD) to express either normal or reduced exercise capacity appears regulated by factors such as fatigue, quality‐of‐life and disorder severity. The associations between physical exercise and risk for PD, the effects of exercise on idiopathic Parkinsonism and quality‐of‐life, the effects of exercise on animal laboratory models of Parkinsonism and dopamine (DA) loss following neurotoxic insults, and the effects of exercise on the DA precursor, l‐Dopa, efficacy are examined. It would appear to be case that in view of the particular responsiveness of the dopaminergic neurons to exercise, the principle of ‘use it or lose’ may be of special applicability among PD patients.</abstract><subject lang="en"><genre>Keywords</genre><topic>Parkinsonism</topic><topic>exercise</topic><topic>quality‐of‐life</topic><topic>function</topic><topic>dopamine</topic></subject><relatedItem type="host"><titleInfo><title>Acta Neurologica Scandinavica</title></titleInfo><genre type="Journal">journal</genre><identifier type="ISSN">0001-6314</identifier><identifier type="eISSN">1600-0404</identifier><identifier type="DOI">10.1111/(ISSN)1600-0404</identifier><identifier type="PublisherID">ANE</identifier><part><date>2011</date><detail type="volume"><caption>vol.</caption><number>123</number></detail><detail type="issue"><caption>no.</caption><number>2</number></detail><extent unit="pages"><start>73</start><end>84</end><total>12</total></extent></part></relatedItem><identifier type="istex">64987335DC08043EBFF4362DAEAA5CCDD0E660FE</identifier><identifier type="DOI">10.1111/j.1600-0404.2010.01360.x</identifier><identifier type="ArticleID">ANE1360</identifier><accessCondition type="use and reproduction" contentType="copyright">© 2010 John Wiley & Sons A/S</accessCondition><recordInfo><recordContentSource>WILEY</recordContentSource><recordOrigin>Blackwell Publishing Ltd</recordOrigin></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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